Skip to content

Tricarboxylic Acid (TCA) Cycle

  • most fuels ultimately enter the TCA cycle when O\(_2\) is present
  • location: mitochondrial matrix

Mitochondrial structure (relevant features)

Outer membrane

  • contains porins: small molecules and ions can move in/out
  • intermembrane space composition is similar to cytoplasm

Inner membrane

  • high protein:lipid ratio (ETC proteins)
  • contains many TIMs (translocase of inner membranes)
  • cristae = folds of inner membrane that increase surface area

Matrix

  • enzyme-rich compartment where TCA enzymes operate

Reaction:

  • pyruvate + CoA-SH → acetyl-CoA + CO\(_2\)

Notes:

  • acetyl group is linked to the -SH group of CoA
  • catalyzed by pyruvate dehydrogenase complex (PDC)
  • pyruvate is transported into the matrix via TIMs

Pyruvate dehydrogenase complex (PDC)

Enzymes and coenzymes :

1) Pyruvate dehydrogenase (E1)

Coenzyme:

  • thiamine pyrophosphate (TPP), vitamin B\(_1\)

Function:

  • converts pyruvate + TPP → hydroxyethyl-TPP + CO\(_2\)

2) Dihydrolipoyl transacetylase (E2)

Coenzymes:

  • lipoamide
  • CoA (vitamin B\(_5\))

Functions:

  • hydroxyethyl-TPP + lipoamide → TPP + acetyl-dihydrolipoamide
  • acetyl-dihydrolipoamide + CoA → acetyl-CoA + fully reduced lipoamide

(then re-oxidation coupling)

  • fully reduced lipoamide + FAD → FADH\(_2\) + lipoamide (via dihydrolipoyl dehydrogenase)

3) Dihydrolipoyl dehydrogenase (E3)

Coenzymes:

  • FAD
  • NAD\(^+\)

Function:

  • FADH\(_2\) + NAD\(^+\) → FAD + NADH

Regulation of PDC

PDC kinase

  • phosphorylates PDC and inactivates it

Allosteric regulation :

  • up: acetyl-CoA, NADH
  • down: ADP, pyruvate

PDC phosphatase

  • removes phosphate and activates PDC

Allosteric regulation :

  • up: Ca\(^{2+}\)

Product inhibition

  • NADH and acetyl-CoA inhibit the complex

The TCA cycle (9-step listing)

1) Citrate synthase

  • acetyl-CoA + oxaloacetate → citrate + CoA-SH (CoA-SH leaves in exchange for water)

Notes:

  • product inhibition (citrate binds active site)
  • citrate slows PFK-1

2) Aconitase

  • citrate ⇌ cis-aconitate (dehydration)
  • cis-aconitate ⇌ isocitrate (hydration)

3) Isocitrate dehydrogenase

  • isocitrate → oxalosuccinate → \(\alpha\)-ketoglutarate + CO\(_2\)
  • NAD\(^+\) → NADH
  • requires proton

Regulation :

  • up: ADP, NAD\(^+\)
  • down: ATP, NADH

4) \(\alpha\)-Ketoglutarate dehydrogenase complex

  • \(\alpha\)-ketoglutarate + CoA-SH + NAD\(^+\) → succinyl-CoA + CO\(_2\) + NADH

Notes :

  • product inhibition by succinyl-CoA and NADH
  • has 3 enzymes:

  • \(\alpha\)-ketoglutarate dehydrogenase

  • dihydrolipoyl succinylase
  • dihydrolipoyl dehydrogenase
  • uses the same coenzymes as PDC

5) Succinate thiokinase

  • succinyl-CoA + P\(_i\) + GDP → succinate + CoA-SH + GTP

6) Succinate dehydrogenase (membrane-associated)

  • succinate + FAD → fumarate + FADH\(_2\)

7) Fumarase

  • fumarate + OH\(^-\) → carbanion transition state → malate (with H\(^+\))

8) Malate dehydrogenase

  • malate + NAD\(^+\) → oxaloacetate + NADH

Note :

  • this is effectively driven forward by coupling to citrate synthase (acetyl-CoA + oxaloacetate is highly exergonic)

Energy output (no ETC accounting beyond NADH/FADH\(_2\) ATP equivalents)

Assume:

  • NADH = 2.5 ATP
  • FADH\(_2\) = 1.5 ATP

Glycolysis (stage 1)

  • 2 NADH, 2 pyruvate, 2 ATP
  • total: 7 ATP equivalents

Pyruvate oxidation (stage 2)

  • 2 NADH
  • total: 5 ATP equivalents

Citric acid cycle (stage 3)

  • 6 NADH, 2 FADH\(_2\), 2 GTP
  • total: 20 ATP equivalents

Anaerobic respiration

  • 2 ATP

Anaplerotic reaction (refilling TCA intermediates)

  • pyruvate + HCO\(_3^-\) + ATP → oxaloacetate enzyme: pyruvate carboxylase

Regulation :

  • acetyl-CoA is an allosteric activator

Coenzyme:

  • biotin, vitamin B\(_7\)

Inhibitors of TCA / linked steps

Sodium fluoroacetate

  • converted to fluorocitrate
  • fluorocitrate inhibits aconitase

Arsenite

  • binds fully reduced lipoamide
  • inhibits PDC and \(\alpha\)-ketoglutarate dehydrogenase complex

Clinical note: PDC deficiency

  • insufficient PDC activity → increased lactate production (lactic acidemia)
  • may be relieved by high-fat, low-carbohydrate diet